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Synaptic Landscape: Oliver Ullrich
Scientist and photographer Oliver Ullrich (b. 1960) returns to explore the Cabo de Gata in Andalusia, one of the driest regions in Europe.At first glance, the landscape here appears frozen and unchanging, but Ullrich tracks down the places where nature seems to communicate.Various natural objects, wind, water, and sun interact and reveal nature here as a living thing that can be experienced as a poetic rather than a purely rational quality.The images in Synaptic Landscape bear witness to vitality, beauty, and change in equilibrium — as signs of a healthy landscape.At the same time, however, they highlight the vulnerability of nature and remind us of the threat posed by humankind. Text in English and German.
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Synaptic Plasticity in Neurodegenerative Disorders
This book explores the pivotal role of synaptic plasticity in the pathogenesis, progression, and potential treatment of neurodegenerative disorders.The initial chapter provides an in-depth understanding of the complexity and impact of neurodegenerative conditions.It discusses the association of mitochondrial dysfunction, epigenetic influences, and neuroinflammation with synaptic plasticity in neurodegenerative diseases.The following chapters review the dynamic changes that occur at the cellular and synaptic levels in Parkinson's disease, Alzheimer's disease, and Huntington's disease, paving the way for innovative therapeutic strategies.Furthermore, the book presents various computational tools and methodologies essential for enhancing our understanding of synaptic plasticity.It examines the transformative role of artificial intelligence tools in addressing synaptic impairment across various neurodegenerative diseases. Discusses the role of synaptic plasticity in neurodegenerative diseases, shedding light on how dynamic changes occur at the cellular and synaptic levelsExplores the transformative role of artificial intelligence tools in addressing synaptic impairment across various neurodegenerative diseasesProvides a comprehensive overview of neurodegenerative disorders, including pathogenesis, etiology, and treatment strategiesPresents tools and techniques used to simulate the complex system biology of synaptic plasticityExamines the role of computational neuroscience in understanding and potentially treating conditions such as multiple sclerosis and amyotrophic lateral sclerosisToward the end, the book explores the role of synaptic impairment and computational neuroscience in understanding and potentially treating conditions such as multiple sclerosis and amyotrophic lateral sclerosis.With its multifaceted approach, this book serves as a useful resource for researchers, clinicians, and students in the fields of neuroscience, computational biology, and neurology.
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Exhaustivity, Contrastivity, and the Semantics of Mandarin Cleft-related Structures
Exhaustivity, Contrastivity, and the Semantics of Mandarin Cleft-related Structures investigates the semantics of the cleft and cleft-related structures in Mandarin, which, over several decades, have presented analytical challenges for semantic theory.The goal of this book, in broad terms, is three-fold: (i) to figure out what clefting adds to the semantics of a sentence; (ii) to set apart the meaning and the discourse function of each type of cleft-related structure; and (iii) to provide a uniform analysis of Mandarin clefts and their related structures.More specifically, it addresses the following questions: (i) what is the semantics of Mandarin clefts? (ii) what do exhaustivity and contrastivity contribute to the meaning of clefts? (iii) what are the semantic (or pragmatic) factors that determine the variation of clefts, related structures, and canonical sentences? and (iv) cross-linguistically speaking, how do Mandarin shi...de cleft and its related structures differ from similar constructions such as English it-cleft, French c’est cleft, and German es-cleft?This book will be informative for linguists who are working on cleft constructions and focus on sensitive structures cross-linguistically, and those interested in experimental semantics and pragmatics.
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Cleft Palate And Craniofacial Conditions: A Comprehensive Guide To Clinical Management
Cleft Palate and Craniofacial Conditions: A Comprehensive Guide to Clinical Management, Fourth Edition is ideal for the graduate course on cleft palate and craniofacial conditions, or as a source book for healthcare professionals who provide services in this area. Updated with full color illustrations and photos, the Fourth Edition of this bestselling title has been reorganized to improve clarity and flow, with new references and information reflecting the most current research in the field.Key features such as Case Studies help the learner understand how to apply key information in a clinical setting, while Speech Notes highlight how anomalies and surgeries affect speech and resonance. As the most comprehensive text in the market, Cleft Palate and Craniofacial Conditions answers the questions of ‘when,’ ‘what,’ ‘why,’ and most importantly ‘how’ when ensuring evaluation results in an accurate diagnosis. Because the author is both an experienced clinician and instructor, this textbook goes beyond the classroom, serving as a very practical “how-to” guide as well as a source of didactic and theoretical information. KEY FEATURESNew full-color design includes 689 photos and illustrationsA complete Glossary of over 650 defined terms Information is summarized in tables for easy learning, including tables of terms for normal and abnormal craniofacial, oral, dental, and pharyngeal structures and anomaliesA robust pedagogy includes Chapter Outlines, illustrations, Case Studies, Speech Notes, definitions, and Discussion Questions295 Clinical Videos illustrate speech samples, evaluation techniques, videofluoroscopy, nasopharyngoscopy, nasometry, and speech therapy techniques, more than any other text in the field!New full-color design includes 689 photos and illustrationsA complete Glossary of over 650 defined terms Information is summarized in tables for easy learning, including tables of terms for normal and abnormal craniofacial, oral, dental, and pharyngeal structures and anomaliesA robust pedagogy includes Chapter Outlines, illustrations, Case Studies, Speech Notes, definitions, and Discussion Questions295 Clinical Videos illustrate speech samples, evaluation techniques, videofluoroscopy, nasopharyngoscopy, nasometry, and speech therapy techniques, more than any other text in the field!
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What is the purpose of the synaptic cleft?
The synaptic cleft is a small gap between the axon terminal of one neuron and the dendrite of another neuron. Its purpose is to ensure that signals are transmitted in one direction only, from the presynaptic neuron to the postsynaptic neuron. This directional flow of information helps prevent confusion and ensures that nerve impulses are transmitted accurately. Additionally, the synaptic cleft allows for the release and diffusion of neurotransmitters, which are essential for communication between neurons.
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How does the impulse cross the synaptic cleft?
The impulse crosses the synaptic cleft through a process called neurotransmission. When the electrical impulse reaches the end of the presynaptic neuron, it triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to specific receptors on the postsynaptic neuron, causing a change in its membrane potential and generating a new electrical impulse. This allows the impulse to continue propagating through the nervous system.
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Is the requested ion flow in the synaptic cleft?
Yes, the requested ion flow occurs in the synaptic cleft. When an action potential reaches the presynaptic neuron, it triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic neuron, causing ion channels to open and allowing specific ions to flow into the postsynaptic neuron. This ion flow is essential for the transmission of signals between neurons and is a key mechanism in synaptic communication.
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How does THC affect dopamine and serotonin in the synaptic cleft?
THC, the active compound in marijuana, affects dopamine and serotonin levels in the synaptic cleft by increasing the release of these neurotransmitters. It does this by binding to cannabinoid receptors in the brain, which then leads to an increase in dopamine and serotonin release. This can result in the feeling of euphoria and relaxation commonly associated with marijuana use. However, chronic use of THC can lead to a decrease in dopamine and serotonin levels, which may contribute to the development of addiction and mood disorders.
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Innovation Competency Model : Shaping Faculty Academic Innovation Development in China's Higher Education
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Piano Keychain Music Instruments Cleft Picture Glass Ball Key Chains Guitar Clarinet Flute Violin
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2Pcs/Lot Dental Tooth Slooth Cleft Crack Detector Probe 135℃ Fracture Locating Crown Bridge Seat
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Rethinking Education for Sustainable Development : Research, Policy and Practice
This book explores how education can be used as a tool to promote sustainability practices as the world faces huge challenges related to climate change and public health.The chapters consider all types of literacy approaches that fall under the umbrella of Education for Sustainable Development (ESD).These approaches include scientific literacy, ecological literacy, health literacy, education on climate change and climate resilience, environmental education and others linking education, global health, and the environment more broadly. “Education” is used in the widest sense to incorporate non-formal, informal and formal/school settings.This volume will help to bring these interconnected areas together and interrogate their research methods, assumptions, field-based application and their policy potential.Taking a critical approach to ESD, the book suggests new pedagogies, tools, and technologies to strengthen the way we educate about sustainability issues and go beyond the current thinking about ESD.The book includes a foreword by Jeffrey Sachs, Director of the Center for Sustainable Development at Columbia University, USA.
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Are there too many neurotransmitters in the synaptic cleft in ADHD?
In ADHD, there is not an excess of neurotransmitters in the synaptic cleft. Instead, there is a dysregulation in the levels of neurotransmitters, particularly dopamine and norepinephrine. These neurotransmitters are not properly released, received, or cleared from the synaptic cleft, leading to difficulties in signal transmission between neurons. This imbalance contributes to the symptoms of ADHD, such as inattention, hyperactivity, and impulsivity.
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Why do neurotransmitters need to be removed quickly from the synaptic cleft?
Neurotransmitters need to be removed quickly from the synaptic cleft to ensure that the signal between neurons is precise and specific. If neurotransmitters linger in the synaptic cleft for too long, they can continue to stimulate the postsynaptic neuron, leading to overstimulation or interference with the next signal. Rapid removal of neurotransmitters also allows for the synapse to reset quickly and be ready for the next signal transmission, ensuring efficient communication between neurons.
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Do people with ADHD have too many neurotransmitters in the synaptic cleft?
No, people with ADHD do not have too many neurotransmitters in the synaptic cleft. In fact, ADHD is often associated with lower levels of certain neurotransmitters, such as dopamine and norepinephrine, in the brain. These lower levels can contribute to difficulties with attention, impulse control, and hyperactivity that are characteristic of ADHD. Treatment for ADHD often involves medications that help increase the levels of these neurotransmitters in the brain to improve symptoms.
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Why do neurotransmitters need to be removed from the synaptic cleft at high speed?
Neurotransmitters need to be removed from the synaptic cleft at high speed to ensure the proper functioning of the nervous system. If neurotransmitters linger in the synaptic cleft for too long, they can continue to stimulate the postsynaptic neuron, leading to overstimulation and potential excitotoxicity. Rapid removal of neurotransmitters helps to prevent continuous signaling and allows for precise control of neuronal communication, ensuring that the nervous system functions efficiently and effectively.
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